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Multi-organ safety in human organoids

Compare normal kidney, lung and GI organoids with adjacent non-tumor liver or other relevant models in a coordinated exposure design to rank organ risks and focus follow-up safety studies.

Bright-field, AT1, AT2, DAPI and merged views of lung organoids under normal and LPS conditionsView image

Normal lung organoidsLPS injury & alveolar phenotypes

Kidney injury-related qPCR readouts after cisplatin exposure in H9-derived kidney organoidsView image

Normal kidney organoidsCisplatin injury & molecular readouts

TRiCBIO normal-lung LPS and kidney organoid cisplatin study data

Lung–kidney safety combination

Compare organ-specific injury in lung and kidney organoids

Lung LPS-injury and kidney cisplatin readouts demonstrate organ-specific safety assays that can be coordinated around candidate risk, exposure, controls and injury endpoints.

Study focus

What this study can help you decide

  1. 01Which organ risks should be compared first?
  2. 02How should organ-specific endpoints be aligned within a comparable exposure design?
  3. 03Which risk signals warrant the next stage of investigation?

STUDY APPROACH

Match the model and assays to your program

Suitable projects

Candidates requiring a comparable exposure design across several organs to rank risks and focus follow-up safety work.

Model strategy

Build a panel of normal kidney, lung and GI models plus adjacent non-tumor liver or other relevant systems around mechanism, expected exposure and known risks, with organ-specific identity and functional QC.

Assay set

Use a comparable exposure and control framework with organ-specific viability, morphology, histology, molecular injury and functional endpoints.

Key controls

Plan dose and timing around the candidate, with exposure conditions, vehicle and positive-injury controls tailored to each organ and QC for each model batch.

How results inform your program

Compare organ-specific signals in exposure context, rank priority risks and select the mechanism, recovery or in vivo studies to run next.

Study workflow

Build the study around priority organ risks

The organs that matter most depend on a candidate's mechanism, expected exposure and known risks of related drugs. TRiCBIO selects normal-tissue models and organ-specific injury and functional endpoints to identify early risk signals and guide the next studies.

01

Risk ranking and model panel

Use target distribution, modality, expected exposure and known risk to prioritize organs and build a model panel matched to each program.

02

Model selection and assay development

Build on kidney drug-injury data and normal-lung LPS studies. Develop additional endpoints in adjacent non-tumor liver, gastric or female-reproductive models from culture and identity profiling; assess IBD models for inflammation or barrier questions.

03

Exposure design and injury readouts

Set dose, timing, controls and sampling windows for each organ, then measure organ-specific responses through morphology, viability, histology, barrier, molecular-injury or functional readouts.

04

Integrated interpretation and study extension

Separate treatment-associated injury from culture and differentiation state, then prioritize added batches, mechanism work or confirmation in another model.

Expected study outputs

What you receive

01

Organ-specific model QC

For each organ model, receive model identity, functional QC results and the injury endpoints used in the study.

02

Results by organ

Receive viability, morphology, histology or molecular injury data alongside each organ's exposure conditions, controls and sampling times, so differences can be interpreted in context.

03

Integrated interpretation of risk signals

A summary of organ-specific risk signals, model performance and candidate-specific differences, with priority organs and follow-up study recommendations.

Relevant model systems

Build the model panel around organ risk

Start with normal kidney, lung, liver and gastric models, then select organs and endpoints around candidate mechanism, exposure and known risks.

Study design

Each organ model uses its own identity QC, injury endpoints and acceptance criteria, with endometrial and fallopian models available through tailored studies. Results support early organ-injury signal identification, candidate comparison and follow-on study design, complementing exposure, in vivo and GLP/regulatory toxicology evidence.

PROJECT DISCUSSION

Plan a risk-directed multi-organ study

Start with the candidate, exposure conditions and organ risks that need priority comparison. Our team will assemble the organ panel and complementary injury endpoints.